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Role of cell defense against oxidative damage in the resistance of Candida albicans to the killing effect of amphotericin B.

A laboratory-derived mutant of Candida albicans B311 (L) and a clinical isolate (C) of C. albicans, both lacking membrane ergosterol, were less susceptible to amphotericin B (AmB)-induced cell membrane permeability to K+ and lethality than was the wild-type laboratory strain (B311) which contained e...

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書誌詳細
出版年:Antimicrob Agents Chemother
主要な著者: Sokol-Anderson, M, Sligh, J E, Elberg, S, Brajtburg, J, Kobayashi, G S, Medoff, G
フォーマット: Artigo
言語:Inglês
出版事項: American Society for Microbiology (ASM) 1988
主題:
オンライン・アクセス:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC172255/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/3293525/
https://ncbi.nlm.nih.govhttps://doi.org/10.1128/aac.32.5.702
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