Role of cell defense against oxidative damage in the resistance of Candida albicans to the killing effect of amphotericin B.
A laboratory-derived mutant of Candida albicans B311 (L) and a clinical isolate (C) of C. albicans, both lacking membrane ergosterol, were less susceptible to amphotericin B (AmB)-induced cell membrane permeability to K+ and lethality than was the wild-type laboratory strain (B311) which contained e...
保存先:
| 出版年: | Antimicrob Agents Chemother |
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| 主要な著者: | , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
American Society for Microbiology (ASM)
1988
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| 主題: | |
| オンライン・アクセス: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC172255/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/3293525/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/aac.32.5.702 |
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