Ciprofloxacin does not inhibit mitochondrial functions but other antibiotics do.
At clinical concentrations, ciprofloxacin did not inhibit mitochondrial DNA replication, oxidative phosphorylation, protein synthesis, or mitochondrial mass (transmembrane potential). No difference in supercoiled forms of DNA was observed. The tetracyclines and chloramphenicol inhibited protein synt...
Uloženo v:
| Vydáno v: | Antimicrob Agents Chemother |
|---|---|
| Hlavní autoři: | , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
American Society for Microbiology (ASM)
1990
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC171543/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/2327755/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/aac.34.1.167 |
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