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Delayed expression of large conductance K(+) channels reshaping agonist-induced currents in mouse pancreatic acinar cells
Epithelial secretory cells display cell-specific mechanisms of fluid secretion and express large conductance voltage- and Ca(2+)-activated K(+) (Maxi-K) channels that generate the membrane negativity for effective Cl(−) exit to the lumen. Rat and mouse pancreatic acinar cells had been thought to be...
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| Hauptverfasser: | , , , , , |
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| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
Blackwell Science Inc
2005
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1665597/ https://ncbi.nlm.nih.gov/pubmed/15611028 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/jphysiol.2004.077834 |
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