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Coagulation defect of congenital tyrosinaemia.

Three infants with hereditary tyrosinaemia had a severe coagulation defect due to a combination of deficient hepatic synthesis of clotting factors and a consumption coagulopathy. In all three plasma failed to clot normally with the venom of Bothrops atrox (Reptilase) and we attribute this to a defec...

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Detalhes bibliográficos
Main Authors: Evans, D I, Sardharwalla, I B
Formato: Artigo
Idioma:Inglês
Publicado em: 1984
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC1628840/
https://ncbi.nlm.nih.gov/pubmed/6508343
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