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Heat shock proteins, end effectors of myocardium ischemic preconditioning?
The purpose of this study was to investigate (1) whether ischemia-reperfusion increased the content of heat shock protein 72 (Hsp72) transcripts and (2) whether myocardial content of Hsp72 is increased by ischemic preconditioning so that they can be considered as end effectors of preconditioning. Tw...
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| Hlavní autoři: | , , , , , , |
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| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
Cell Stress Society International
2006
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1576469/ https://ncbi.nlm.nih.gov/pubmed/17009598 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1379/CSC-181R1.1 |
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