The pore-forming subunit of the K(ATP) channel is an important molecular target for LPS-induced vascular hyporeactivity in vitro

1. ATP-sensitive K(+) (K(ATP)) channel activation is implicated in the vascular hyporeactivity occurring in septic shock. However, channel inhibition with the sulphonylurea receptor (SUR) antagonist, glibenclamide (Glib) fails to reverse lipopolysaccharide (LPS)-induced vascular hyporeactivity in vi...

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Autores principales: O'Brien, Alastair J, Thakur, Gita, Buckley, James F, Singer, Mervyn, Clapp, Lucie H
Formato: Artigo
Lenguaje:Inglês
Publicado: 2005
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Papers
Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC1576013/
https://ncbi.nlm.nih.gov/pubmed/15655519
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/sj.bjp.0706065
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