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Inhibition of kinin breakdown prolongs retention and action of bradykinin in a myocardial B(2) receptor compartment
1. The high efficacy of ACE inhibitors to potentiate the actions of kinins might be explained by a hypothetical compartment in which B(2)-receptors are colocalized with kinin degrading enzymes. To demonstrate the functional consequence of such a compartment we compared the myocardial uptake and the...
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| Autori principali: | , , , , |
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| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
2003
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1573665/ https://ncbi.nlm.nih.gov/pubmed/12540521 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/sj.bjp.0705036 |
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