Bacterial lipopolysaccharide induces long-lasting IgA deficiency concurrently with features of polyclonal B cell activation in normal and in lupus-prone mice.

Podobne zapisy

• Bacterial lipopolysaccharide enhances deposition of immune complexes and exacerbates nephritis in BXSB lupus-prone mice.
  od: Granholm, N A, i wsp.
  Wydane: (1991)
• Bacterial lipopolysaccharide transforms mesangial into proliferative lupus nephritis without interfering with processing of pathogenic immune complexes in NZB/W mice.
  od: Cavallo, T., i wsp.
  Wydane: (1990)
• Pathogenic role of polyclonal and polymeric IgA in a murine model of mesangial proliferative glomerulonephritis with IgA deposition.
  od: Muso, E, i wsp.
  Wydane: (1991)
• Lipopolysaccharide from gram-negative bacteria enhances polyclonal B cell activation and exacerbates nephritis in MRL/lpr mice.
  od: Cavallo, T, i wsp.
  Wydane: (1990)
• Repeated exposure to bacterial lipopolysaccharide interferes with disposal of pathogenic immune complexes in mice.
  od: Cavallo, T, i wsp.
  Wydane: (1990)