Ectopic expression of E47 or E12 promotes the death of E2A-deficient lymphomas
Mice with null mutations in the E2A gene are highly susceptible to the spontaneous development of thymic lymphomas. To understand better how E2A deficiency may contribute to lymphomagenesis, we have observed the consequences of enforced expression of the E2A gene products E12 and E47 in cell lines d...
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| Publicado no: | Proc Natl Acad Sci U S A |
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| Principais autores: | , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
National Academy of Sciences
1999
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC15339/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/9927682/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.96.3.996 |
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