טוען...
Increased vascular permeability in C1 inhibitor–deficient mice mediated by the bradykinin type 2 receptor
Heterozygosity for C1 inhibitor (C1INH) deficiency results in hereditary angioedema. Disruption of the C1INH gene by gene trapping enabled the generation of homozygous- and heterozygous-deficient mice. Mating of heterozygous-deficient mice resulted in the expected 1:2:1 ratio of wild-type, heterozyg...
שמור ב:
| Main Authors: | , , , , |
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| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
American Society for Clinical Investigation
2002
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC150945/ https://ncbi.nlm.nih.gov/pubmed/11956243 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI14211 |
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