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The polycystin-1 C-terminal fragment triggers branching morphogenesis and migration of tubular kidney epithelial cells
Mutations of either PKD1 or PKD2 cause autosomal dominant polycystic kidney disease, a syndrome characterized by extensive formation of renal cysts and progressive renal failure. Homozygous deletion of Pkd1 or Pkd2, the genes encoding polycystin-1 and polycystin-2, disrupt normal renal tubular diffe...
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| Main Authors: | , , , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
American Society for Clinical Investigation
2002
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC150870/ https://ncbi.nlm.nih.gov/pubmed/11854320 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI12867 |
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