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Thrombin-activatable fibrinolysis inhibitor (TAFI) deficiency is compatible with murine life
To investigate the consequence of deficiency in thrombin-activatable fibrinolysis inhibitor (TAFI), we generated homozygous TAFI-deficient mice by targeted gene disruption. Intercrossing of heterozygous TAFI mice produced offspring in the expected Mendelian ratio, indicating that transmission of the...
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| 主要な著者: | , , , , , , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
American Society for Clinical Investigation
2002
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC150816/ https://ncbi.nlm.nih.gov/pubmed/11781355 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI12119 |
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