Further evidence that accelerated muscle protein breakdown during sepsis is not mediated by prostaglandin E2.

Prostaglandin E2 (PGE2) reportedly increases protein break-down in skeletal muscle. The role of PGE2 for accelerated muscle proteolysis during sepsis, however, is controversial. In this study, the effect of the prostaglandin synthesis inhibitor indomethacin on PGE2 release and protein breakdown in s...

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Detalhes bibliográficos
Main Authors: Hasselgren, P O, Warner, B W, Hummel, R P, James, J H, Ogle, C K, Fischer, J E
Formato: Artigo
Idioma:Inglês
Publicado em: 1988
Assuntos:
Research Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC1493441/
https://ncbi.nlm.nih.gov/pubmed/3162671
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