Increased proinflammatory endothelial response to S100A8/A9 after preactivation through advanced glycation end products

BACKGROUND: Atherosclerosis is an inflammatory disease in which a perpetuated activation of NFkappaB via the RAGE (receptor for advanced glycation end products)-MAPK signalling pathway may play an important pathogenetic role. As recently S100 proteins have been identified as ligands of RAGE, we soug...

Πλήρης περιγραφή

Αποθηκεύτηκε σε:
Λεπτομέρειες βιβλιογραφικής εγγραφής
Κύριοι συγγραφείς: Ehlermann, Philipp, Eggers, Kai, Bierhaus, Angelika, Most, Patrick, Weichenhan, Dieter, Greten, Johannes, Nawroth, Peter P, Katus, Hugo A, Remppis, Andrew
Μορφή: Artigo
Γλώσσα:Inglês
Έκδοση: BioMed Central 2006
Θέματα:
Original Investigation
Διαθέσιμο Online:https://ncbi.nlm.nih.gov/pmc/articles/PMC1475836/
https://ncbi.nlm.nih.gov/pubmed/16573830
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1475-2840-5-6
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