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Smad3 deficiency alters key structural elements of the extracellular matrix and mechanotransduction of wound closure
The loss of TGFβ or its downstream mediator, Smad3, key players in tissue repair, accelerates closure of incisional wounds in mice. In contrast, we now report that excisional ear wounds in mice lacking Smad3 enlarge compared with wild-type controls resulting from changes in extracellular matrix mole...
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| Main Authors: | , , , , , , , , |
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| 格式: | Artigo |
| 語言: | Inglês |
| 出版: |
National Academy of Sciences
2006
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| 主題: | |
| 在線閱讀: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1474013/ https://ncbi.nlm.nih.gov/pubmed/16754864 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0602473103 |
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