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An enzymatic ruler modulates Lewis antigen glycosylation of Helicobacter pylori LPS during persistent infection

Helicobacter pylori persistently colonizes about half the human population and contributes to the development of peptic ulcer disease and gastric cancer. This organism has evolved means to structurally alter its surface characteristics to evade innate and adaptive immune responses. H. pylori produce...

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Detalles Bibliográficos
Main Authors: Nilsson, Christina, Skoglund, Anna, Moran, Anthony P., Annuk, Heidi, Engstrand, Lars, Normark, Staffan
Formato: Artigo
Idioma:Inglês
Publicado: National Academy of Sciences 2006
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Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC1413829/
https://ncbi.nlm.nih.gov/pubmed/16477004
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0511119103
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