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An enzymatic ruler modulates Lewis antigen glycosylation of Helicobacter pylori LPS during persistent infection

Helicobacter pylori persistently colonizes about half the human population and contributes to the development of peptic ulcer disease and gastric cancer. This organism has evolved means to structurally alter its surface characteristics to evade innate and adaptive immune responses. H. pylori produce...

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Autori principali: Nilsson, Christina, Skoglund, Anna, Moran, Anthony P., Annuk, Heidi, Engstrand, Lars, Normark, Staffan
Natura: Artigo
Lingua:Inglês
Pubblicazione: National Academy of Sciences 2006
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC1413829/
https://ncbi.nlm.nih.gov/pubmed/16477004
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0511119103
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