The Amino-Terminal Extensions of the Longer Sendai Virus C Proteins Modulate pY701-Stat1 and Bulk Stat1 Levels Independently of Interferon Signaling

The Sendai virus (SeV) C proteins are known to interact with Stat1 to prevent interferon (IFN)-induced pY701-Stat1 formation and IFN signaling. Nevertheless, pY701-Stat1 levels paradoxically increase during SeV infection. The C proteins also induce bulk Stat1 instability in some cells, similar to ru...

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Vydáno v:	J Virol
Hlavní autoři:	Garcin, Dominique, Marq, Jean-Baptiste, Goodbourn, Stephen, Kolakofsky, Daniel
Médium:	Artigo
Jazyk:	Inglês
Vydáno:	American Society for Microbiology (ASM) 2003
Témata:	Virus-Cell Interactions
On-line přístup:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC141115/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/12551969/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/JVI.77.4.2321-2329.2003
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