Point Mutations in Herpes Simplex Virus Type 1 oriL, but Not in oriS, Reduce Pathogenesis during Acute Infection of Mice and Impair Reactivation from Latency

Gelijkaardige items

• Site-Directed Mutagenesis of Large DNA Palindromes: Construction and In Vitro Characterization of Herpes Simplex Virus Type 1 Mutants Containing Point Mutations That Eliminate the oriL or oriS Initiation Function
  door: Balliet, John W., et al.
  Gepubliceerd in: (2005)
• Cloning and characterization of herpes simplex virus type 1 oriL: comparison of replication and protein-DNA complex formation by oriL and oriS.
  door: Hardwicke, M A, et al.
  Gepubliceerd in: (1995)
• Herpes simplex virus type 1 oriL is not required for virus replication or for the establishment and reactivation of latent infection in mice.
  door: Polvino-Bodnar, M, et al.
  Gepubliceerd in: (1987)
• Evidence that the Herpes Simplex Virus Type 1 ICP0 Protein Does Not Initiate Reactivation from Latency In Vivo
  door: Thompson, R. L., et al.
  Gepubliceerd in: (2006)
• Herpes Simplex Virus Latency-Associated Transcript Sequence Downstream of the Promoter Influences Type-Specific Reactivation and Viral Neurotropism
  door: Bertke, Andrea S., et al.
  Gepubliceerd in: (2007)