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A linear lattice model for polyglutamine in CAG-expansion diseases
Huntington's disease and several other neurological diseases are caused by expanded polyglutamine [poly(Gln)] tracts in different proteins. Mechanisms for expanded (>36 Gln residues) poly(Gln) toxicity include the formation of aggregates that recruit and sequester essential cellular proteins...
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| Main Authors: | , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
The National Academy of Sciences
2002
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC129321/ https://ncbi.nlm.nih.gov/pubmed/12193654 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.182393899 |
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