Molecular basis for the dichotomy in Plasmodium falciparum adhesion to CD36 and chondroitin sulfate A
Plasmodium falciparum-infected erythrocytes adhere dichotomously to the host receptors CD36 and chondroitin sulfate A (CSA). This dichotomy is associated with parasite sequestration to microvasculature beds (CD36) or placenta (CSA), leading to site-specific pathogenesis. Both properties are mediated...
保存先:
| 出版年: | Proc Natl Acad Sci U S A |
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| 主要な著者: | , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
National Academy of Sciences
2002
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC126617/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/12096191/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.152321599 |
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