Signaling disrupts mSin3A binding to the Mad1-like Sin3-interacting domain of TIEG2, an Sp1-like repressor

A Sin3-interacting domain (SID) originally described in Mad proteins is necessary for both transcriptional repression and growth suppression by these transcription factors. We recently reported that a structurally and functionally related Mad1-like SID is also present in five Sp1-like repressor prot...

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Detalhes bibliográficos

Publicado no:	EMBO J
Principais autores:	Ellenrieder, Volker, Zhang, Jin-San, Kaczynski, Joanna, Urrutia, Raul
Formato:	Artigo
Idioma:	Inglês
Publicado em:	Nature Publishing Group 2002
Assuntos:	Article
Acesso em linha:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC126002/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/12006497/ https://ncbi.nlm.nih.govhttps://doi.org/10.1093/emboj/21.10.2451
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