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PI 3-kinase gamma and protein kinase C-zeta mediate RAS-independent activation of MAP kinase by a Gi protein-coupled receptor.

Receptors coupled to the inhibitory G protein Gi, such as that for lysophosphatidic acid (LPA), have been shown to activate MAP kinase through a RAS-dependent pathway. However, LPA (but not insulin) has now been shown to activate MAP kinase in a RAS-independent manner in CHO cells that overexpress a...

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Autors principals: Takeda, H, Matozaki, T, Takada, T, Noguchi, T, Yamao, T, Tsuda, M, Ochi, F, Fukunaga, K, Inagaki, K, Kasuga, M
Format: Artigo
Idioma:Inglês
Publicat: 1999
Matèries:
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC1171133/
https://ncbi.nlm.nih.gov/pubmed/9889195
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/emboj/18.2.386
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