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Mutations in both the structured domain and N-terminus of histone H2B bypass the requirement for Swi-Snf in yeast.
The chromatin elements targeted by the ATPdependent, Swi-Snf nucleosome-remodeling complex are unknown. To address this question, we generated mutations in yeast histone H2B that suppress phenotypes associated with the absence of Swi-Snf. Sin- (Swi-Snf-independent) mutations occur in residues involv...
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| Autores principales: | , |
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| Formato: | Artigo |
| Lenguaje: | Inglês |
| Publicado: |
1999
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| Materias: | |
| Acceso en línea: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1171117/ https://ncbi.nlm.nih.gov/pubmed/9878065 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/emboj/18.1.229 |
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