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Activation of the JNK pathway is essential for transformation by the Met oncogene.
The Met/Hepatocyte Growth Factor (HGF) receptor tyrosine kinase is oncogenically activated through a rearrangement that creates a hybrid gene Tpr-Met. The resultant chimeric p65(Tpr-Met) protein is constitutively phosphorylated on tyrosine residues in vivo and associates with a number of SH2-contain...
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| Autores principales: | , , |
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| Formato: | Artigo |
| Lenguaje: | Inglês |
| Publicado: |
1997
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| Materias: | |
| Acceso en línea: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1169874/ https://ncbi.nlm.nih.gov/pubmed/9184210 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/emboj/16.10.2634 |
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