NIH-3T3 cells transformed with a ras oncogene exhibit a protein kinase C-mediated inhibition of agonist-stimulated Ca2+ inflow.

Documenti analoghi

• Inhibition of Ca2+ inflow causes an abrupt cessation of growth-factor-induced repetitive free Ca2+ transients in single NIH-3T3 cells.
  di: Polverino, A J, et al.
  Pubblicazione: (1991)
• NIH-3T3 cells transformed by the EJ-ras oncogene exhibit reduced platelet-derived growth factor-mediated Ca2+ mobilization.
  di: Benjamin, C W, et al.
  Pubblicazione: (1988)
• Resistance to oncogenic transformation in revertant R1 of human ras-transformed NIH 3T3 cells.
  di: Kuzumaki, N, et al.
  Pubblicazione: (1989)
• The stimulation by sodium fluoride of plasma-membrane Ca2+ inflow in isolated hepatocytes. Evidence that a GTP-binding regulatory protein is involved in the hormonal stimulation of Ca2+ inflow.
  di: Hughes, B P, et al.
  Pubblicazione: (1987)
• Isolation of recessive (mediator-) revertants from NIH 3T3 cells transformed with a c-H-ras oncogene.
  di: Yamada, H, et al.
  Pubblicazione: (1990)