Regulation of hepatic fructose 2,6-bisphosphate concentrations and lipogenesis after re-feeding in euthyroid and hyperthyroid rats. A regulatory role for glycogenesis.

Eitemau Tebyg

• Effects of nutritional status and acute variation in substrate supply on cardiac and skeletal-muscle fructose 2,6-bisphosphate concentrations.
  gan: French, T J, et al.
  Cyhoeddwyd: (1988)
• Continued glucose output after re-feeding contributes to glucose intolerance in hyperthyroidism.
  gan: Holness, M J, et al.
  Cyhoeddwyd: (1987)
• The disposition of carbohydrate between glycogenesis, lipogenesis and oxidation in liver during the starved-to-fed transition.
  gan: Holness, M J, et al.
  Cyhoeddwyd: (1988)
• Hepatic carbon flux after re-feeding. Hyperthyroidism blocks glycogen synthesis and the suppression of glucose output observed in response to carbohydrate re-feeding.
  gan: Holness, M J, et al.
  Cyhoeddwyd: (1987)
• The relationship between changes in lipid fuel availability and tissue fructose 2,6-bisphosphate concentrations and pyruvate dehydrogenase complex activities in the fed state.
  gan: French, T J, et al.
  Cyhoeddwyd: (1988)