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Hepatic carbon flux after re-feeding in the glycogen-storage-disease (gsd/gsd) rat.

In this study we utilized the phosphorylase b kinase-deficient (gsd/gsd) rat as a model of hepatic substrate utilization where there is a constraint on glycogenesis imposed by the maintenance of high glycogen concentrations. Glucose re-feeding of 48 h-starved gsd/gsd rats led to suppression of hepat...

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Dettagli Bibliografici
Autori principali: Holness, M J, Palmer, T N, Worrall, E B, Sugden, M C
Natura: Artigo
Lingua:Inglês
Pubblicazione: 1987
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC1148646/
https://ncbi.nlm.nih.gov/pubmed/3481265
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