טוען...

Protein kinase C-dependent and -independent mechanisms regulating the parotid substance P receptor as revealed by differential effects of protein kinase C inhibitors.

Substance P-induced inositol trisphosphate (InsP3) formation was inhibited by 1 microM-4 beta-phorbol 12,13-dibutyrate (PDBu) in rat parotid acinar cells. The inhibitory effect of PDBu was reversed by the protein kinase C inhibitors H-7 or K252a. Substance P also elicits a persistent desensitization...

תיאור מלא

שמור ב:
מידע ביבליוגרפי
Main Authors: Sugiya, H, Putney, J W
פורמט: Artigo
שפה:Inglês
יצא לאור: 1988
נושאים:
גישה מקוונת:https://ncbi.nlm.nih.gov/pmc/articles/PMC1135464/
https://ncbi.nlm.nih.gov/pubmed/2464997
תגים: הוספת תג
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