Inhibition by pertussis toxin of the activation of Na(+)-dependent uridine transport in dimethyl-sulphoxide-induced HL-60 leukaemia cells.

समान संसाधन

• Enhancement of pertussis-toxin-sensitive Na(+)-dependent uridine transporter activity in HL-60 granulocytes by N-formylmethionyl-leucyl-phenylalanine.
  द्वारा: Goh, L B, और अन्य
  प्रकाशित: (1993)
• Induction of the differentiation of HL-60 cells by phorbol 12-myristate 13-acetate activates a Na(+)-dependent uridine-transport system. Involvement of protein kinase C.
  द्वारा: Lee, C W, और अन्य
  प्रकाशित: (1991)
• Treatment of HL-60 cells with dimethyl sulphoxide inhibits the formation of beta-N-acetylhexosaminidase S.
  द्वारा: Emiliani, C, और अन्य
  प्रकाशित: (1990)
• Decrease in equilibrative uridine transport during monocytic differentiation of HL-60 leukaemia: involvement of protein kinase C.
  द्वारा: Lee, C W
  प्रकाशित: (1994)
• Prostaglandin-concentration-dependent desensitization of adenylate cyclase in human erythroleukaemia (HEL) cells is abolished by pertussis toxin and enhanced by induction by dimethyl sulphoxide.
  द्वारा: Ashby, B, और अन्य
  प्रकाशित: (1991)