Involvement of the Zinc-Binding Capacity of Sendai Virus V Protein in Viral Pathogenesis
The V protein of Sendai virus (SeV) is nonessential to virus replication in cell culture but indispensable to viral pathogenicity in mice. The highly conserved cysteine-rich zinc finger-like domain in its carboxyl terminus is believed to be responsible for this viral pathogenicity. In the present st...
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| 出版年: | J Virol |
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| 主要な著者: | , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
American Society for Microbiology (ASM)
2000
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| 主題: | |
| オンライン・アクセス: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC112313/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/10933690/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jvi.74.17.7834-7841.2000 |
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