Lack of S-Adenosylmethionine Results in a Cell Division Defect in Escherichia coli
The enzyme S-adenosylmethionine (SAM) synthetase, the Escherichia coli metK gene product, produces SAM, the cell’s major methyl donor. We show here that SAM synthetase activity is induced by leucine and repressed by Lrp, the leucine-responsive regulatory protein. When SAM synthetase activity falls b...
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| 出版年: | J Bacteriol |
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| 主要な著者: | , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
American Society for Microbiology (ASM)
1998
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| 主題: | |
| オンライン・アクセス: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC107330/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/9658005/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jb.180.14.3614-3619.1998 |
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