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Abnormalities in the biosynthesis of thromboxane A2 and prostacyclin in children with cyanotic congenital heart disease.

BACKGROUND--Children with cyanotic congenital heart disease and pulmonary outflow tract obstruction have shortened platelet survival times and are susceptible to thrombosis and organ infarction. Thromboxane A2 and prostacyclin have opposing actions on platelet aggregability and an imbalance in their...

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Detalhes bibliográficos
Publicado no:Br Heart J
Main Authors: Adatia, I, Barrow, S E, Stratton, P, Ritter, J M, Haworth, S G
Formato: Artigo
Idioma:Inglês
Publicado em: BMJ Publishing Group 1993
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC1024947/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8435245/
https://ncbi.nlm.nih.govhttps://doi.org/10.1136/hrt.69.2.179
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