SAPAP3 regulates epileptic seizures involving GluN2A in post-synaptic densities

Abstract Aberrantly synchronized neuronal discharges in the brain lead to epilepsy, a devastating neurological disease whose pathogenesis and mechanism are unclear. SAPAP3, a cytoskeletal protein expressed at high levels in the postsynaptic density (PSD) of excitatory synapses, has been well studied...

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Bibliografiset tiedot
Päätekijät: Yanke Zhang, Junhong Wu, Yin Yan, Yixue Gu, Yuanlin Ma, Min Wang, Hui Zhang, Kaiyan Tao, Yang Lü, Weihua Yu, Wei Jing, Xuefeng Wang, Xin Tian
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Nature Publishing Group 2022-05-01
Sarja:Cell Death and Disease
Linkit:https://doi.org/10.1038/s41419-022-04876-9
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