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Human cytomegalovirus UL97 kinase prevents the deposition of mutant protein aggregates in cellular models of Huntington's disease and Ataxia

The presence of aggregates of abnormally expanded polyglutamine (polyQ)-containing proteins are a pathological hallmark of a number of neurodegenerative diseases including Huntington's disease (HD) and spinocerebellar ataxia-3 (SCA3). Previous studies in cellular, Drosophila, and mouse models of HD...

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Principais autores: Cristy Tower, Lianwu Fu, Rachel Gill, Mark Prichard, Mathieu Lesort, Elizabeth Sztul
Formato: Artigo
Idioma:Inglês
Publicado em: Elsevier 2011-01-01
coleção:Neurobiology of Disease
Assuntos:
UL97
Polyglutamine
Aggresome
Ataxin 3
Huntingtin
PML bodies
Acesso em linha:http://www.sciencedirect.com/science/article/pii/S096999611000272X
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