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Ferroptosis and inflammation are modulated by the NFIL3-ACSL4 axis in sepsis associated-acute kidney injury

Abstract Sepsis-associated acute kidney injury (SA-AKI) increases the risk of death in patients with sepsis, and its major pathological change is the death of renal tubular cells. However, the mechanism of its occurrence remains unclear. Sepsis can lead to circadian dysregulation, and the rhythm gen...

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Wedi'i Gadw mewn:
Manylion Llyfryddiaeth
Prif Awduron: Zhong Xiao, Jie Zhang, Zhimin Qiu, Hongbing Liu, Hua Ding, Hi Li, Yuanxin Liu, Xiaohua Zou, Juan Long
Fformat: Artigo
Iaith:Inglês
Cyhoeddwyd: Nature Publishing Group 2024-08-01
Cyfres:Cell Death Discovery
Mynediad Ar-lein:https://doi.org/10.1038/s41420-024-02113-0
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