Prostate cancer cell proliferation is influenced by LDL-cholesterol availability and cholesteryl ester turnover

Abstract Background Prostate cancer growth is driven by androgen receptor signaling, and advanced disease is initially treatable by depleting circulating androgens. However, prostate cancer cells inevitably adapt, resulting in disease relapse with incurable castrate-resistant prostate cancer. Androg...

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Autori principali:	Nikki L. Raftopulos, Tinashe C. Washaya, Andreas Niederprüm, Antonia Egert, Mariam F. Hakeem-Sanni, Bianca Varney, Atqiya Aishah, Mariya L. Georgieva, Ellinor Olsson, Diandra Z. dos Santos, Zeyad D. Nassar, Blake J. Cochran, Shilpa R. Nagarajan, Meghna S. Kakani, Jordan F. Hastings, David R. Croucher, Kerry-Anne Rye, Lisa M. Butler, Thomas Grewal, Andrew J. Hoy
Natura:	Artigo
Lingua:	Inglês
Pubblicazione:	BMC 2022-01-01
Serie:	Cancer & Metabolism
Soggetti:	Prostate cancer LDL LDL-cholesterol Cell proliferation Cholesteryl ester ACAT1
Accesso online:	https://doi.org/10.1186/s40170-021-00278-1
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