RIPK3-deficient mice were not protected from nephrotoxic nephritis
Abstract Background Necrotizing glomerular lesions are a feature of severe glomerulonephritis. Unlike apoptosis, cellular necrosis has the potential to release damage-associated proteins into the microenvironment, thereby potentiating inflammation. Until recently necrosis was thought to be an unregu...
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| Huvudupphov: | , , , |
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| Materialtyp: | Artigo |
| Språk: | Inglês |
| Utgiven: |
BMC
2018-03-01
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| Serie: | BMC Nephrology |
| Ämnen: | |
| Länkar: | http://link.springer.com/article/10.1186/s12882-018-0850-4 |
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