Human myocardial mitochondrial oxidative capacity is impaired in mild acute heart transplant rejection

Abstract Aims Acute cellular rejection (ACR) following heart transplantation (HTX) is associated with long‐term graft loss and increased mortality. Disturbed mitochondrial bioenergetics have been identified as pathophysiological drivers in heart failure, but their role in ACR remains unclear. We aim...

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Detalhes bibliográficos

Principais autores:	Daniel Scheiber, Elric Zweck, Sophie Albermann, Tomas Jelenik, Maximilian Spieker, Florian Bönner, Patrick Horn, Heinz‐Peter Schultheiss, Ganna Aleshcheva, Felicitas Escher, Udo Boeken, Payam Akhyari, Michael Roden, Malte Kelm, Julia Szendroedi, Ralf Westenfeld
Formato:	Artigo
Idioma:	Inglês
Publicado em:	Oxford University Press 2021-12-01
coleção:	ESC Heart Failure
Assuntos:	Acute cellular heart transplant rejection Mitochondrial respiration Myocardial inflammation Heart failure
Acesso em linha:	https://doi.org/10.1002/ehf2.13607
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