Physiological febrile heat stress increases cytoadhesion through increased protein trafficking of Plasmodium falciparum surface proteins into the red blood cell

Fever is a hallmark of malaria. Several studies have linked febrile temperatures to reduced parasite viability, but also to increased cytoadhesion, a key driver of pathology. However, different mechanisms have been proposed to cause changes in cytoadhesion and parasite sensitivity to heat. Here, we...

Täydet tiedot

Bibliografiset tiedot

Päätekijät:	David Jones, Hugo Belda, Malgorzata Broncel, Gwendolin Fuchs, David Anaguano, Stephanie D Nofal, Moritz Treeck
Aineistotyyppi:	Artigo
Kieli:	Inglês
Julkaistu:	eLife Sciences Publications Ltd 2026-05-01
Sarja:	eLife
Aiheet:	Plasmodium falciparum fever pathogenesis protein trafficking malaria
Linkit:	https://elifesciences.org/articles/107860
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