SZC-6 Promotes Diabetic Wound Healing in Mice by Modulating the M1/M2 Macrophage Ratio and Inhibiting the MyD88/NF-χB Pathway

<b>Background/Objectives</b>: The prolonged M1-like pro-inflammatory polarization of macrophages is a key factor in the delayed healing of diabetic ulcers (DU). SIRT3, a primary mitochondrial deacetylase, has been identified as a regulator of inflammation and represents a promising new therapeutic t...

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Auteurs principaux:	Ang Xuan, Meng Liu, Lingli Zhang, Guoqing Lu, Hao Liu, Lishan Zheng, Juan Shen, Yong Zou, Shengyao Zhi
Format:	Artigo
Langue:	Inglês
Publié:	MDPI AG 2025-07-01
Collection:	Pharmaceuticals
Sujets:	diabetic SZC-6 macrophages wound healing
Accès en ligne:	https://www.mdpi.com/1424-8247/18/8/1143
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