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O-GlcNAcylation-induced GSK-3β activation deteriorates pressure overload-induced heart failure via lack of compensatory cardiac hypertrophy in mice

O-GlcNAc transferase (OGT) modulates many functions of proteins via O-GlcNAcylation that adds O-linked β-N-acetylglucosamine (O-GlcNAc) to the serine/threonine residues of proteins. However, the role of O-GlcNAcylation in cardiac remodeling and function is not fully understood. To examine the effect...

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Principais autores: Mahito Matsuno, Shunichi Yokoe, Takehiro Nagatsuka, Hirofumi Morihara, Kazumasa Moriwaki, Michio Asahi
פורמט: Artigo
שפה:Inglês
יצא לאור: Frontiers Media S.A. 2023-03-01
סדרה:Frontiers in Endocrinology
נושאים:
O-GlcNAcylation
heart failure
hypertrophy
transverse aortic constriction (TAC)
GSK-3β
גישה מקוונת:https://www.frontiersin.org/articles/10.3389/fendo.2023.1122125/full
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