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CaMKII Metaplasticity Drives Aβ Oligomer-Mediated Synaptotoxicity

Summary: Alzheimer’s disease (AD) is emerging as a synaptopathology driven by metaplasticity. Indeed, reminiscent of metaplasticity, oligomeric forms of the amyloid-β peptide (oAβ) prevent induction of long-term potentiation (LTP) via the prior activation of GluN2B-containing NMDA receptors (NMDARs)...

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Autors principals: Patricio Opazo, Silvia Viana da Silva, Mario Carta, Christelle Breillat, Steven J. Coultrap, Dolors Grillo-Bosch, Matthieu Sainlos, Françoise Coussen, K. Ulrich Bayer, Christophe Mulle, Daniel Choquet
Format: Artigo
Idioma:Inglês
Publicat: Elsevier 2018-06-01
Col·lecció:Cell Reports
Accés en línia:http://www.sciencedirect.com/science/article/pii/S2211124718307824
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