QR-koodi

Exercise attenuates polyglutamine‐mediated neuromuscular degeneration in a mouse model of spinal and bulbar muscular atrophy

Abstract Background Spinal and bulbar muscular atrophy (SBMA) is a hereditary neuromuscular disorder caused by the expansion of trinucleotide cytosine–adenine–guanine (CAG) repeats, which encodes a polyglutamine (polyQ) tract in the androgen receptor (AR) gene. Recent evidence suggests that, in addi...

Täydet tiedot

Tallennettuna:
Päätekijät: Tomoki Hirunagi, Hideaki Nakatsuji, Kentaro Sahashi, Mikiyasu Yamamoto, Madoka Iida, Genki Tohnai, Naohide Kondo, Shinichiro Yamada, Ayuka Murakami, Seiya Noda, Hiroaki Adachi, Gen Sobue, Masahisa Katsuno
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Wiley 2024-02-01
Sarja:Journal of Cachexia, Sarcopenia and Muscle
Aiheet:
AMPK
exercise
polyglutamine disease
spinal and bulbar muscular atrophy
Linkit:https://doi.org/10.1002/jcsm.13344
Tagit: Lisää tagi
Ei tageja, Lisää ensimmäinen tagi!

Samankaltaisia teoksia