TRPC4 deletion elicits behavioral defects in sociability by dysregulating expression of microRNA-138-2

Summary: To investigate whether the defects in transient receptor potential canonical 4 (TRPC4), which is strongly expressed in the hippocampus, are implicated in ASD, we examined the social behaviors of mice in which Trpc4 was deleted (Trpc4−/−). Trpc4−/− mice displayed the core symptoms of ASD, na...

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Detalhes bibliográficos

Principais autores:	Jee Young Seo, Hye-Ryeong Jo, Seung Hoon Lee, Do Gyeong Kim, Huiju Lee, Ye Lim Kim, Young In Choi, Sung Jun Jung, Hyeon Son
Formato:	Artigo
Idioma:	Inglês
Publicado em:	Elsevier 2024-01-01
coleção:	iScience
Assuntos:	Behavioral neuroscience Molecular neuroscience Neuroscience Rodent behavior
Acesso em linha:	http://www.sciencedirect.com/science/article/pii/S2589004223026949
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