Circuitry and Synaptic Dysfunction in Alzheimer’s Disease: A New Tau Hypothesis
For more than five decades, the field of Alzheimer’s disease (AD) has focused on two main hypotheses positing amyloid-beta (Aβ) and Tau phosphorylation (pTau) as key pathogenic mediators. In line with these canonical hypotheses, several groups around the world have shown that the synaptotoxicity in...
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| Huvudupphov: | , , |
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| Materialtyp: | Artigo |
| Språk: | Inglês |
| Utgiven: |
Wiley
2020-01-01
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| Serie: | Neural Plasticity |
| Länkar: | http://dx.doi.org/10.1155/2020/2960343 |
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