β-catenin regulates muscle glucose transport via actin remodelling and M-cadherin binding

Objective: Skeletal muscle glucose disposal following a meal is mediated through insulin-stimulated movement of the GLUT4-containing vesicles to the cell surface. The highly conserved scaffold-protein β-catenin is an emerging regulator of vesicle trafficking in other tissues. Here, we investigated t...

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Hlavní autoři:	Stewart W.C. Masson, Brie Sorrenson, Peter R. Shepherd, Troy L. Merry
Médium:	Artigo
Jazyk:	Inglês
Vydáno:	Elsevier 2020-12-01
Edice:	Molecular Metabolism
Témata:	Beta-catenin Actin-remodelling Glucose transport M-cadherin GLUT4 trafficking
On-line přístup:	http://www.sciencedirect.com/science/article/pii/S2212877820301654
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