A multi-state model of the CaMKII dodecamer suggests a role for calmodulin in maintenance of autophosphorylation.
Ca2+/calmodulin-dependent protein kinase II (CaMKII) accounts for up to 2 percent of all brain protein and is essential to memory function. CaMKII activity is known to regulate dynamic shifts in the size and signaling strength of neuronal connections, a process known as synaptic plasticity. Increasi...
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| Asıl Yazarlar: | , , , , , , , |
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| Materyal Türü: | Artigo |
| Dil: | Inglês |
| Baskı/Yayın Bilgisi: |
Public Library of Science (PLoS)
2019-12-01
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| Seri Bilgileri: | PLoS Computational Biology |
| Online Erişim: | https://journals.plos.org/ploscompbiol/article/file?id=10.1371/journal.pcbi.1006941&type=printable |
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