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The Distinct Function of p21Waf1/Cip1 With p16Ink4a in Modulating Aging Phenotypes of Werner Syndrome by Affecting Tissue Homeostasis

Human Werner syndrome (WS) is an autosomal recessive progeria disease. A mouse model of WS manifests the disease through telomere dysfunction-induced aging phenotypes, which might result from cell cycle control and cellular senescence. Both p21Waf1/Cip1 (p21, encoded by the Cdkn1a gene) and p16Ink4a...

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Principais autores: Yongjin Zhang, Chihao Shao, Haili Li, Kun Wu, Lixin Gong, Quan Zheng, Juhua Dan, Shuting Jia, Xiaodan Tang, Xiaoming Wu, Ying Luo
Formato: Artigo
Idioma:Inglês
Publicado em: Frontiers Media S.A. 2021-02-01
coleção:Frontiers in Genetics
Assuntos:
p21Waf1/Cip1
p16INK4a
aging
tissue homeostasis
Werner syndrome
Acesso em linha:https://www.frontiersin.org/articles/10.3389/fgene.2021.597566/full
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