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S100B Impairs Oligodendrogenesis and Myelin Repair Following Demyelination Through RAGE Engagement

Increased expression of S100B and its specific receptor for advanced glycation end products (RAGE) has been described in patients with multiple sclerosis (MS), being associated with an active demyelinating process. We previously showed that a direct neutralization of S100B reduces lysophosphatidylch...

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Detalhes bibliográficos
Principais autores: Gisela Santos, Andreia Barateiro, Dora Brites, Adelaide Fernandes
Formato: Artigo
Idioma:Inglês
Publicado em: Frontiers Media S.A. 2020-09-01
coleção:Frontiers in Cellular Neuroscience
Assuntos:
Acesso em linha:https://www.frontiersin.org/article/10.3389/fncel.2020.00279/full
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