KLC3 drives gastric cancer progression by stabilizing SLC2A5 to activate MAPK signaling and promote epithelial-mesenchymal transition
Abstract Background Gastric cancer (GC) is associated with poor prognosis due to its intricate molecular mechanisms that drive multiple malignant functions. While KLC3 has been found to be abnormally expressed in various tumors, its specific mechanistic role in GC remains largely unknown. Aims This...
I tiakina i:
| Ngā kaituhi matua: | , , , , , , |
|---|---|
| Hōputu: | Artigo |
| Reo: | Inglês |
| I whakaputaina: |
BMC
2025-11-01
|
| Rangatū: | BMC Cancer |
| Ngā marau: | |
| Urunga tuihono: | https://doi.org/10.1186/s12885-025-15084-x |
| Ngā Tūtohu: |
Kāore He Tūtohu, Me noho koe te mea tuatahi ki te tūtohu i tēnei pūkete!
|